Community Acquired Pneumonia
A specific etiological diagnosis is often difficult or impossible to make in community-acquired pneumonia. Given this, the true incidence of each microbial etiology is unknown. In an otherwise healthy host, probably 95 to 99 per cent of community-acquired pneumonias are due to viral, mycoplasma, pneumococcal (Streptococcus pneumoniae) or Legionella infections, and some authors estimate that viruses and mycoplasma account for more than half. In patients with underlying disease, other organisms may be found which fortunately tend to follow a certain predictable pattern: Streptococcus pneumoniae, Staphylococcus aureus, and Haemophilus influenzae are commonly responsible for bacterial pneumonia following influenza; S. pneumoniae and H. influenzae in patients with chronic bronchitis; and anaerobes in patients with pneumonia following aspiration of oropharyngeal contents. Highly resistant gram-negative bacilli are very rarely responsible for community-acquired pneumonia.
If pneumonia is suspected, a physical examination is essential; however, even in the absence of abnormal physical signs, such as crackles or evidence of consolidation, pneumonia may be present. A chest x-ray is required to differentiate pneumonia (infiltrate expected) from bronchitis. While certain radiological patterns have been associated with different organisms, such associations are of a nonspecific nature and are of little value in an individual patient. Serology may be obtained if viral, mycoplasma, or Legionella infection is suspected but does not aid immediate clinical decisions because a delay is incurred in demonstrating a rising titer (up to five weeks). Sputum cultures are also of limited value. Sensitivity is only 50 per cent for pneumococci, and other fastidious organisms are difficult to culture or are overgrown by oral flora. If positive, cultures of blood or pleural fluid provide a precise etiological diagnosis, but negative results are much more common. Sputum Gram’s stain remains the time-honored approach, but some patients are unable to expectorate, and even when sputum is obtained, it is usually of poor quality and contaminated by oral flora 75 per cent of the time. When an optimal specimen is obtained with extreme care, there is still a 38 per cent false-negative rate for pneumococcal pneumonia. Invasive techniques to obtain selective cultures are rarely required in community-acquired pneumonia.
TheraPV is commonly instituted on an empirical basis because of the limited information provided by the above diagnostic approaches. One suggested approach is depicted in Figure 23-1. In the normal host, a viral etiology is suggested by the presence of a dry cough and systemic illness jn a young person. A bacterial infection is commoner in adult patients, who often present suddenly with rigors, productive cough, chest pain, and a high white cell count. Patients with Legionella infection have features of both. Erythromycin is the most practical empirical agent, as it is the drug of choice for mycoplasma and Legionella infection and is also useful in pneumococcal pneumonia. The antibiotic selected for an abnormal host depends on the specific circumstances . The decision of whether to hospitalize the patient is often based on the patient’s underlying disease status.
- POSTCAPILLARY PULMONARY HYPERTENSION
- RESPIRATORY CONTROL CENTERS
- Community Acquired Pneumonia
- Aspiration Pneumonia and Lung Abscess
- ANGINA PECTORIS
- MANAGEMENT OF CARDIAC ARRHYTHMIAS
- ARTERIAL TRAUMA
- Mechanism of Proteinuria
- EFFECTORS OF THE RESPIRATORY SYSTEM
- VENTRICULAR RHYTHM DISTURBANCES
- Therapy
- ADAPTATION TO NEPHRON LOSS
- NAUSEA AND VOMITING
- Alterations in Glomerular Hemodynamics, Parathyroid Hormone Metabolism, and Systemic Arterial Blood Pressure
- Incidence
- The Use of Diuretics
- Pathogenic Mechanisms - Mechanism of Injury
- NONMEDICAL MANAGEMENT OF ANGINA PECTORIS
- Multiple Myeloma
- Other Cystic Diseases
- Indications for Dialysis and Adequacy of Dialysis
- CLINICAL MANIFESTATIONS OF MALABSORPTION
- Pulmonary Vasculitis
- MYOCARDIAL DISEASE - MYOCARDITIS
- CHRONIC RENAL FAILURE
- Alterations in Drug Doses in Patients with Renal Failure
- GLOMERULAR DISEASE
- MECHANISMS OF ARRHYTHMOGENESIS
- MEDIASTINAL DISEASE
- Renal Artery Stenosis
- MICROSCOPIC ANATOMY
- Renal Tumors
- LABORATORY TESTS TOR BILIRUBIN
- Pneumonia in the Immunocompromised Host
- CLINICAL CLASSIFICATION OF JAUNDICE