POSTCAPILLARY PULMONARY HYPERTENSION
This results from the passive effects of an elevated left atrial pressure due to mitral valve disease or prolonged left ventricular failure. If it persists, pulmonary artery pressure gradually increases above that explained by passive transmission of the elevated outflow pressure. Initially this increase is due to reflex vasoconstriction, but eventually vascular remodeling occurs. This additional contribution to pulmonary hypertension can be quantified by the increasing gradient between the pulmonary diastolic pressure and the left atrial pressure.
- Proliferative Glomerulonephritis
- Diagnosis
- ATRIAL RHYTHM DISTURBANCES
- NORMAL ESOPHAGEAL PHYSIOLOGY
- RENAL PHARMACOLOGY
- EFFECTS OF PULMONARY HYPERTENSION ON PULMONARY FUNCTION
- PHYSIOLOGY OF THE CORONARY CIRCULATION
- Amyloidosis
- PHYSIOLOGY OF THE SYSTEMIC CIRCULATION
- HEMATOLOGY
- THE AIRWAY STRUCTURE
- Mesangioproliferative Glomerulonephritis
- Reduction in GFR
- Phenytoin
- Clinical Course, Pathogenesis, and Anatomy of Acute Tubular Necrosis
- Determination of Kidney Anatomy and Renal Blood Flow
- ARTERIOSCLEROSIS OBLITERANS
- Familial Polyposis of the Colon
- Treatment
- EMPHYSEMA
- OBLITERATIVE OR OBSTRUCTIVE PULMONARY HYPERTENSION
- Other Clearly Extrinsic Causes of Diffuse Infiltrative Lung Disease
- Treatment and Prognosis
- Metabolism of Drugs in Patients with Renal Insufficiency
- PHYSIOLOGY OF THE PULMONARY CIRCULATION
- HYPERKINETIC PULMONARY HYPERTENSION
- Alberto N. v. Hawkins
- THE COMMON CLINICAL MANIFESTATIONS OF GASTROINTESTINAL DISEASE
- Clinical Manifestations
- Phosphate Balance
- Blood Chemistries
- Liddle’s Syndrome
- Sigmoidoscopy and Colonoscopy
- PHYSICAL THERAPY AND REHABILITATION
- Aminoaciduria